The Spectrum of Neurologic Disease in the Severe Acute Respiratory Syndrome Coronavirus 2 Pandemic Infection

The Spectrum of Neurologic Disease in the Severe Acute Respiratory Syndrome Coronavirus 2 Pandemic Infection

MD: Article reprenant les troubles neurologiques post covid: "SARS that appeared in patients 2 to 3 weeks into the course of the illness, mainly consisting of either an axonal peripheral neuropathy or a myopathy with elevated creatinine kinase.4 At the time, it was unclear whether some of these manifestations might be owing to critical illness–related effects,5 but pathology subsequently showed that patients with SARS had widespread vasculitis seen in many organs, including striated muscle,6 suggesting that the clinical features in these neuromuscular patients might be more than just nonspecific complications of severe illness." 5 of 206 patients with SARS in Singapore developed large-vessel strokes. Four of these patients had their strokes in the setting of critical illness owing to SARS, and 3 were associated with significant episodes of hypotension.

Coagulopathy and Antiphospholipid Antibodies in Patients with Covid-19

Coagulopathy and Antiphospholipid Antibodies in Patients with Covid-19

MD: Cas rapportés d un homme de 69 ans atteint de Covid-19 diagnostiqué en janvier 2020 à Wuhan, ainsi que deux autres patients avec forme grave de Covid-19 pec dans la même unité de soins intensifs. Une coagulopathie et des anticorps antiphospholipides ont été observés chez les trois patients.

Potential Effects of Coronaviruses on the Cardiovascular System A Review - JAMA

Potential Effects of Coronaviruses on the Cardiovascular System A Review - JAMA

IMPORTANCE Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), which causes coronavirus disease 2019 (COVID-19) has reached a pandemic level. Coronaviruses are known to affect the cardiovascular system. We review the basics of coronaviruses, with a focus on COVID-19, along with their effects on the cardiovascular system.

The neuroinvasive potential of SARS‐CoV2 may be at least partially responsible for the respiratory failure of COVID‐19 patients

The neuroinvasive potential of SARS‐CoV2 may be at least partially responsible for the respiratory failure of COVID‐19 patients

Following the severe acute respiratory syndrome coronavirus (SARS‐CoV) and Middle East respiratory syndrome coronavirus (MERS‐CoV), another highly patho- genic coronavirus named SARS‐CoV‐2 (previously known as 2019‐nCoV) emerged in December 2019 in Wuhan, China, and rapidly spreads around the world. This virus shares highly homological sequence with SARS‐CoV, and causes acute, highly lethal pneumonia coronavirus disease 2019 (COVID‐19) with clinical symptoms similar to those reported for SARS‐CoV and MERS‐CoV. The most characteristic symptom of patients with COVID‐19 is respiratory distress, and most of the patients admitted to the intensive care could not breathe spontaneously. Additionally, some patients with COVID‐19 also showed neurologic signs, such as headache, nausea, and vomiting. Increasing evidence shows that coronaviruses are not always confined to the re- spiratory tract and that they may also invade the central nervous system inducing neurological…